Timothy Sellati
4 votes
CD14 Modulates PI3K/AKT/p38-MAPK Licensing of Negative Regulators of TLR Signaling to Restrain Chronic Inflammation
Current thinking emphasizes the primacy of CD14 in facilitating TLR recognition of microbes to initiate proinflammatory signaling events and the importance of p38-MAPK in augmenting such responses….
Received 24 June 2008 18:09 UTC; Posted 25 June 2008
Posted to: Immunology, Microbiology
